Specifically, we anticipated stronger reactivity of participants in the negative 30).īased on our theoretical predictions, we did not expect mood effect onĬardiovascular or electrodermal reactivity during the mood inductions but during mood 95).Ĥ Further analysis revealed that for SBP this disengagement concerned specifically participants in a negative mood who showed stronger SBP reactivity during the first minute of the task (M = 7.46, SE = 1.89) comparing to the second minute (M = 1.88, SE = 2.14), t(12) = 2.29, p. Showed high internal consistency (average Cronbach’s Alpha. Period, because the reactivity scores of the physiological parameters were stable and For the mood inductions, we considered the whole 4 Considering this, ourįurther analyses focused exclusively on the first minute of the mood regulation task during Indicates disengagement of the participants after the first minute. Minutes of the mood regulation task, both ts > 2.00, ps. Focused comparisons found that these main effects wereĬaused by significant decreases in SBP and SCL reactivity between the first and the second Resource-related measures-during the mood regulation task, both Fs > 3.96, ps. Significant time main effects on the reactivity of both SBP and SCL-two central Preliminary ANOVAs of these change scores revealed Reactivity scores for SCL and facial EMG wereĭetermined in the same fashion. Ironson, & Schneiderman, 1991) were computed for each participant by subtracting theīaseline values from the values obtained during the mood induction period and the valuesĪssessed during the mood regulation task. For the cardiovascular measures, change (delta) scores (Llabre, Spitzer, Saab,
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